• Meet the 2018-2019 Executive Council
  • Click HERE to see why the Shock Society is so important to our members.
  • Fall 2018 Newsletter

Welcome to the US Shock Society!  

The mission of the Shock Society is to improve the care of victims of trauma, shock, and sepsis, through:

  • Promoting clinically relevant research into the basic biology of trauma, shock, and sepsis
  • Providing a multidisciplinary forum to integrate and disseminate new knowledge in trauma, shock, and sepsis
  • Promoting the education and mentoring of the next generation of investigators in the field of trauma, shock, and sepsis


Shock Society 2019 Annual Conference - June 8-11, 2019
(Co-located with the 39th SIS Annual Meeting, June 5-8, 2019)

We are excited to announce that in response to the membership's request, the Shock Society and the Surgical Infection Society (SIS) will be having a co-located meeting in 2019. The leadership from both societies have jointly agreed on an exceptional venue at Loews Coronado Bay Resort.
Click HERE to see additional information about this exciting meeting

Membership benefits:

Direct Access to professionals and students sharing your specialty.

Access to Journal, SHOCK®: Injury, Inflammation, and Sepsis.

Award Eligibility

What's New in SHOCK, December 2018?

As 2018 draws to a close, this issue of Shock offers another 17 excellent reports on the Basic Science Aspects of shock, injury, inflammation, and sepsis. An important theme this month is related to the role of the heart in shock-related injury. As more patients survive severe sepsis, the recovery phase after clearance of the infection is of great importance. Cardiac failure is a common finding in sepsis that potentiates the onset of shock. Crowell et al. (1) investigated the different regulation of protein balance in the heart during the acute and recovery phases of sepsis in a mouse cecal ligation and puncture (CLP) model. They found that the acute phase was characterized by proteostasis with increased apoptosis, inflammasome activation and autophagy. During recovery, protein synthesis was increased along with pyroptosis via the noncanonical pathway. . ; Read more...

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